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Parasitology

خانواده كليات
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S.bovis در روده بند و باب نشخواركنندگان اهلي و وحشي و اسب و شتر . (در خوزستان)
S.japonicum در روده بند و باب نشخواركنندگان اهلي و وحشي ، گوشتخواران و جوندگان .
بيماريزاترين گونه ها در گوسفند و گاو.
ُ
S.nasalis در سياهرگهاي مخاط بيني نشخواركنندگان و اسب .
S.haematobium در سرخرگهاي مزانتر ، مثانه ، پروستات و رحم انسان . (در خوزستان)
S.mansoni در سياهرگهاي روده بند انسان (افريقا)
سيرتكاملي :
جفتگيري بين نرو ماده صورت مي گيرد.تخمگذاري ماده ها درعروق و فرورفتن خاردر انتيماي رگ وعدم ورود در گردش خون محيطي . تخم ها از جدار رگ عبور كرده و وارد روده شده و حين خروج با مدفوع تكميل مي شوند .پس از تماس مدفوع با آب در دماي 28 (مطلوبترين دما) ميراسيديوم از تخم خارج مي شود .(7 روز براي تشكيل ميراسيديوم).حلزونهاي آبي ، ميزبان واسط هستند .در ايران براي شيستوزوما بويس:
Bulinus truncatus
در بدن حلزون دونسل اسپوروسيست در بافتهاي نرم، سركر (فوركوسركوس) كه فعالانه از حلزون خارج شده و در آب شنا مي كند و فعالانه از راه پوست بدن و با كمك ترشحات غدد راسي و هضم بافتها وارد ميزبان نهايي( بعضي ها از طريق آب وارد دستگاه گوارش) . در بدن ميزبان تبديل به Schistosomula خون ريه كبد.

بيماريزايي:


اهميت بيماريزايي در گوسفند نسبت به گاو بيشتر.
فرم حاد
:(Intestinal .syn) 9-7 هفته بعد از آلودگي رخ داده و بعلت عبور تعداد زياد تخم از مخاط روده رخ مي دهد . جراحات خونريزي در مخاط روده مخصوصا در خلف روده باريك و سكوم .اسهال و كم اشتهايي. بعلت واكنش آماسي و كانونهاي گرانولوماتوز (به قطر 10-1 ميلي متر)ايجاد شده در اثر تخمگذاري انگل در عروق مزانتر و نفوذ بعدي تخم است .در آلودگي شديد تلف شدن سريع حيوان ممكنست ديده شود .
كرم بالغ باعث
Phlebitis در عروق مزانتر و پروليفراسيون لومن مي شود . در شيستوزوما بويس كرم هاي بالغ بيشتر درعروق مزانتر. با مهاجرت كرم از كبد به روده و تخم گذاري در كبد علائم كم مي شوند .
فرم مزمن
(Hepatic .syn): يك واكنش ايمني با واسطه سلولي كه در حقيقت پاسخ ايمني كبد ميزبان به تخم انگل است .

علائم :
سرفه موقت به علت مهاجرت شيستوزوماي جوان به ريه ، اسهال آبكي ، دهيدراتاسيون ، ضعف، كم خوني و كم شدن آلبومين( به علت خونريزي مخاطي ، كاهش خونسازي و ازدياد حجم پلاسما) و خيز . كاهش فرآورده هاي دامي .مزمن : لاغري ، كم خوني ، كاهش آلبومين خون .

تشخيص :
اسهال ، كم خوني و سابقه خوردن آب از مخازن حاوي ميزبان واسط به تشخيص كمك مي كند.با فاسيوليازيس بابد تفريق داد (اسهال طولاني آغشته به خون).آزمايش مدفوع در مراحل اوليه بعد از بلوغ كرم .بهترين روش كالبد گشائي است .
كالبدگشائي:
مخاطات روده خاكستري ، ضخيم و خيزدار و حاوي كانون هاي خونريزي ، گرانولوما و آماس در اثر تخم كرم . در مقاطع بافتي كبد گرانولوما بر اثر تخم كرم و فيبروز ناشي از تخم ديده مي شود .
درمان :
داروي
Praziquantel داروي انتخابي در شيستوزوميازيس انساني است . 60 mg/Kg در يك دز واحد و يا 20 mg/Kg در سه نوبت به فاصله چهار ساعت .

در انسان Niridazole داروي موثري است اما احتياج به كاربرد آن به مدت 10-5 روز دارد .
در دام:
براي درمان از داروهاي انساني استفاده مي شود .پرازيكانتل داروي انتخابي است .
Trichloorophone(Neguvon) : نتايج متفاوتي از كاربرد آن گزارش شده است . در گاو و گوسفند معمولا 40-30 mg/Kg در سه نوبت استفاده مي كنند و در بعضي از موارد ايجاد مسموميت گزارش شده است .
پيشگيري :
دو فاكتور: الف - درمان حيوانات مبتلا ب- از بين بردن يا از دسترس خارج كردن حلزونهاي ميزبان واسط .
روشها : 1- مبارزه بيولوژيك و شيميايي با حلزونها .
2- جلوگيري از تماس حيوان و انسان با آب آلوده .( نرده كشي و..)
3- از بين بردن حلزونها در مخازن آب.
4- عدم استفاده از كود انساني براي مزارع و يا انباشت آن قبل از مصرف .
5- استفاده از چكمه و لباس مخصوص براي افرادي كه در آب كار مي كنند .
6- ماليدن دور كننده ها
(repellants)مانند Dibutylphtalate و Benzyl benzoite به روي پوست قبل از كار در آب .


ادامه مطلب
+ نوشته شده در  دوشنبه بیست و هشتم فروردین 1391ساعت 5:24 PM  توسط امین احمدی   | 


Description:

The horse roundworm, an ascarid, is a rigid, heavy-bodied worm up to 50 centimeterslong.


Predilection site: Small intestine.

Geographic distribution : Very common throughout the US.



Life cycle:

P. equorum (ascarid; roundworm) is a common nematode parasite which occurs in the small intestine of immature horses world-wide. Adult female ascarids lay eggs in the small intestine, and these eggs pass into the environment within the feces of the host. The infective stage is a larvated egg (containing a second stage larva [L]3); development requires approximately 10 days at temperatures of 25°C to 35°C . Larvated eggs survive in the environment for up to five or 10 years, and infection is acquired through inadvertent ingestion of eggs. Larvae emerge from eggs within the alimentary tract of a horse, and migrate through the liver and lungs before returning to the small intestine approximately one month later as fourth stage larvae (L4). Ascarids mature progressively in the small intestine and achieve patency about 75 to 80 days after infection
P. equorum is one of the rare nematodes which induces absolute acquired immunity. Most horses become immune during the first year of life, so patent ascarid infections are rarely diagnosed in horses over two years of age.





ادامه مطلب
+ نوشته شده در  جمعه بیست و یکم بهمن 1390ساعت 11:38 AM  توسط امین احمدی   | 

بررسی میزان آلودگی به انگل های خارجی در ماهيان كپور پرورشي (Cyprinus carpio) شهرستان مشهد

امین احمدی1*،منصورابراهیمی1، سعیدیغفوری1،کیمیا غفاری2

شرط اول پیشگیری از بروز بیماریها بویژه بیماری های انگلی در ماهیان پرورشی تشخیص انگلهای اختصاصی و غیر اختصاصی ماهیان درمنابع آبی است ،آگاهی کامل از عوامل انگلی کمک شایانی به پیشگیری از بروز بیماری های انگلی می کند.در طی سه ده گذشته بهره برداری شیلات به منظور افزایش تولید ماهی و اشتغال در ایران توسعه یافته است که به بدنبال این افزایش جمعیت، بررسی شیوع بیماری در منابع آبی ، مطالعات انگل شناسی و بیماریهای ناشی از انگل هادرکشور لازم و ضروری به نظر می رسد.نمونه گیری در زمستان(1389) و بهار(1390)  انجام شد. 75 عدد ماهي از مراکز پرورش ماهی صید و در مجاورت یخ به آزمایشگاه انگل شناسی منتقل شدند. مطالعات انگل شناسي با بررسي ضايعات ماكروسكوپي پوست، باله ها و آبشش ماهيان بوسيله استرئوميكروسكوپ و نمونه برداري ازاین نواحی به وسیله گسترش مرطوب صورت گرفت.برای مشاهده انگل های مونوژن آبشش هاراخارج کرده و با کمک استرئوميكروسكوپ گونه مونوژن ساکن درآبشش ها به وسیله ی پیپت پاستور جداسازی شد و بر اساس کلید های تشخیصی، انگل های جداشده تشخیص داده شدند.انگل های جدا شده از 75ماهی کپور شامل: 5.3% لرنه آ از پوست، 4% اکتیو فتیریوس مولتی فیلیس از پوست،2.6% تریکودینا از پوست وآبشش و13.3% گونه های داکتیلوژیروس ازآبشش می باشد.بطور معمول در مزارع پرورش ماهی و یا آبهای ساکن درجه حرارت آب به مقدار زیادی متاثر از درجه حرارت هوا بوده و دامنه تغییرات حرارتی به حدی است که منجر به شیوع برخی انگل ها در تابستان و حذف یا کاهش تعداد در زمستان می شود ، که شیوع کرمهای مونوژن در پوست وآبشش ماهیان ماهیت فصلی دارد. در بررسی رابطه فصل با شیوع انگل، شیوع داکتیلوژیروس ها دراوایل زمستان و اواخر بهار از نظر آماری ارتباط معنی داری داشت.بروز لرنئا در پوست ماهیان دارای ماهیتی فصلی است ودر بهار به اوج خود می رسد، در این بررسی بیشترین میزان الودگی به لرنئا مربوط به فصل بهار بود.انگلهای یافت شده تا حد زیادی دارای چرخه زندگی مستقیم هستند وحضور تک یاخته گانی مانند تریکودینا و ایکتیوفتریوس، ناشی ازافزایش تراکم ماهی وافزایش غنای مواد آلی در آب می باشد، که هشداری جدی برای شیلات و پرورش ماهی است
کلمات کلیدی : انگل، ماهی،کپور،مشهد
ادامه مطلب ....

ادامه مطلب
+ نوشته شده در  جمعه بیست و پنجم آذر 1390ساعت 6:35 PM  توسط امین احمدی   | 

Scabies is caused by a tiny mite called Sarcoptes scabiei. It gets its name from the Latin word scabere which means to scratch. Sarcoptes scabiei is a skin parasite causing severe itching and infections. There are at least 300 million cases every year worldwide. All ages of men and women especially in crowded and unhygienic conditions are at risk.
Sarcoptes scabiei goes through four stages in its life cycle: egg, larva, nymph and adult. The life cycle starts, when an adult female gets in contact with your skin. It crawls to crevices such as elbows, feet, fingers and genital area. It penetrates the skin and burrows a tunnel. It can slice skin with its sharp front legs and mouthparts. The other legs it uses for holding on to the skin with suckers on each leg. It has eight legs in total. It takes about 30 minutes for it to burrow into the skin. It then continues to drill horizontally across the skin laying eggs along the way. The tunnels are usually shaped in a zigzag on the skin surface. It lays 2–3 eggs per day for two months. Then it dies. Six-legged larvae hatch from the eggs within a few days. They find hair follicles where they feed and molt into eight-legged nymphs. In order for the nymph to become an adult male it molts once. This takes about ten days. To become a female it has to molt twice which takes about 17 days. Since the nymph has more time to eat and grow the females are larger than males. Female Sarcoptes scabiei is 0.25–0.35 mm wide and 0.30–0.45 mm long. Males are about half of that size. Mature female and male mate only once. The sperm keep the female fertile for the whole two months that it lays eggs. Males do not usually burrow into the skin but only crawl and feed on it.

ادامه مطلب
+ نوشته شده در  سه شنبه هفدهم آبان 1390ساعت 10:57 AM  توسط امین احمدی   | 

he current importance of Dermanyssus gallinae, mainly in egg layer, in Europe is pointed out. The limits of conventional control methods are underlined (development of mite resistance – future insecticide legislation and animal welfare legislation). Alternative control methods such as feeding deterrents or biological control, are reviewed in the context of poultry pest management.
This parasite has been known for a long time. Its role as a vector of several significant diseases has been emphasized many times. As early as Smith et al. (1944)isolated St Louis encephalitis viruschicken mites. Since then several authors showed its relationship to different poultrychicken pox virus, newcastle virus, agent of pullorum disease and fowl typhoid, and the agent of fowl cholera (see Zeman et al., 1982). However, D. gallinae is most important as a direct pest because it is an obligatory blood-sucking parasite and reproduces very rapidly. from pathogens such as fowl spirochaetosis,
Massive infestations are very common in France with huge numbers of parasites on the birds' cages and on the conveyor belts for eggs. D. gallinae causes irritation, anaemia, and in some cases even death, and may result in blood-stained eggs. The new animal welfare legislation will certainly point out this pathology.
Life cycle of Dermanyssus gallinae
The cycle was first described by Wood (1917). The red mite resembles the northern fowl miteOrnithonyssus sylviarum) in both size and appearance, but its life cycle differs in that the red mitebirds. Red mites only parasitize birds in darkness in order to feed. In farms with battery–cage systems mites are hidden under the conveyor belts of eggs and cage supports. In farms with slatted flooring systems, mites are hidden under the rods, in nest boxes, beneath troughs and in small cracks and crevices in the poultry house walls. ( spends the most of its life away from the
Nymphs and females suck blood while males do so very occasionally and larvae do not feed. Dermanyssus stays on the birds for only 0.5–1.5 h to feed.
Adults mate soon after molting. After fertilization the females begin depositing eggs within about 12 h after feeding. They will feed and deposit eggs several times. Each female is able to lay about 30 eggs in its lifetime.
The egg is small (400×270 μ) oval, smooth and pearly white. Under warm conditions (28–30°C) a larva emerges in 2–3 days. The young larva is white, has 6 legs, and moves about slowly and after 1 day molts to the protonymph stage without feeding. The protonymph has 8 legs; it feeds and molts to a deutonymph which feeds again before becoming an adult ♂ or ♀. In optimal conditions the life cycle (egg-to-egg) can be completed within 7 days


Red mites may live up to 8 months away from poultry.They resist desiccation but cannot tolerate high humidity.

+ نوشته شده در  سه شنبه سوم آبان 1390ساعت 3:16 PM  توسط امین احمدی   | 

Causal Agent

Several protozoan species in the genus Entamoeba colonize humans, but not all of them are associated with disease.  Entamoeba histolytica is well recognized as a pathogenic ameba, associated with intestinal and extraintestinal infections.  The other species are important because they may be confused with E. histolytica in diagnostic investigations.

Life cycle of Entamoeba histolytica


Cysts and trophozoites are passed in feces .  Cysts are typically found in formed stool, whereas trophozoites are typically found in diarrheal stool.  Infection by Entamoeba histolytica occurs by ingestion of mature cysts in fecally contaminated food, water, or hands.  Excystation occurs in the small intestine and trophozoites are released, which migrate to the large intestine.  The trophozoites multiply by binary fission and produce cysts , and both stages are passed in the feces .  Because of the protection conferred by their walls, the cysts can survive days to weeks in the external environment and are responsible for transmission.  Trophozoites passed in the stool are rapidly destroyed once outside the body, and if ingested would not survive exposure to the gastric environment.  In many cases, the trophozoites remain confined to the intestinal lumen ( : noninvasive infection) of individuals who are asymptomatic carriers, passing cysts in their stool.  In some patients the trophozoites invade the intestinal mucosa ( : intestinal disease), or, through the bloodstream, extraintestinal sites such as the liver, brain, and lungs ( : extraintestinal disease), with resultant pathologic manifestations.  It has been established that the invasive and noninvasive forms represent two separate species, respectively E. histolytica and E. dispar.  These two species are morphologically indistinguishable unless E. histolytica is observed with ingested red blood cells (erythrophagocystosis).  Transmission can also occur through exposure to fecal matter during sexual contact (in which case not only cysts, but also trophozoites could prove infective).

ادامه مطلب....


ادامه مطلب
+ نوشته شده در  جمعه بیست و نهم مهر 1390ساعت 2:15 PM  توسط امین احمدی   | 

Diphyllobothrium latum, the fish tapeworm, is the biggest tapeworm in humans. It causes a parasitic infection called diphyllobothriasis which is acquired by eating raw fish infected with the parasite. Diphyllobothriasis is found in Chile, Peru, Uganda and in the Northern Hemisphere (northern Asia, Europe and America) in areas of rivers and lakes. Diphyllobothrium species include: D. cordatum, D. dalliae, D. dendriticum, D. lanceolatum, D. latum, D. pacificum, D. ursi and D. yonagoensis. Diphyllobothrium latum is the most common and mostly found in Scandinavia, the Baltics and western Russia.

The life cycle of Diphyllobothrium latum starts, when immature eggs are passed in the feces of an infected human. The eggs mature in water within three weeks and form oncospheres. Larvae called coracidia hatch and get eaten by freshwater crustaceans such as copepod. After ingestion coracidia develop into procercoid larvae. If the copepod is eaten by a small fish (second intermediate host), the procercoid larvae penetrate the gut and migrate to muscle tissue where they develop into plerocercoid larvae (sparganum), the infective stage for humans. Usually a third intermediate host is needed because humans do not usually eat raw fish this small. If a trout, walleyed pike or perch eats the smaller fish, the plerocercoid larvae once again penetrate the gut and migrate to fish flesh. If a human eats the infected fish raw or undercooked the plerocercoid larvae develop into adults in the small intestine. The adults attach to the intestinal mucosa with two shallow, bilateral grooves (bothria) of their scolex. The scolex is 3 mm long and 1 mm wide. The long, flat body consists of segments, proglottids, that are produced by the neck. Full grown proglottids are about 10 mm wide and 3 mm long. The segments absorb nutrients and have their own digestive systems. As proglottids mature, they release eggs and eventually break off from the body. A Diphyllobothrium latum proglottid is characterized by a rosette-shaped uterus at its center. The eggs are ellipsoidal or oval measuring 55–75 µm by 40–50 µm. They are passed in the feces unembryonated (immature). From the start of the infection it takes about six weeks for the eggs to appear in the feces. One adult tapeworm can shed up to a million eggs per day. It can grow over 10 meters long and live up to 20 years.

 


ادامه مطلب
+ نوشته شده در  جمعه بیست و نهم مهر 1390ساعت 1:30 PM  توسط امین احمدی   | 

 

استرتاژیا _ کرم قهوه ایی شیردان

اسپیکولهای قهوه ایی قطور و پررنگ و کوتاه و 2 یا 3 شاخه

شیردان نشتخوارکنندگان

 


استرونژیلوس ولگاریس _  استرونگل های بزرگ

تشخیص بر اساس ساختار دهان که دارای مجرای پشتی و یک زوج دندان گوشی شکل در کف دهان می باشد

در کولون اسب

 

 

 

 


اسکرجابینما اویس _ کرم سنجاقی گوسفند

مری در قسمت انتهایی واجد یک بولب متسع و کروی است

روده کور گوسفند و بز

 

 

 


اکسیروس اکوئی

مری در ابتدا پهن بوده سپس باریک می شود و در ادامه حالت بولب پیدا میکند

روده بزرگ تک سمی ها

 

 

 

ادامه مطلب ....

 


ادامه مطلب
+ نوشته شده در  پنجشنبه بیست و هشتم مهر 1390ساعت 1:44 PM  توسط راستیانی  | 

Goat Animations

Cryptosporidium


Eimeria spp. (coccidian)

Dictyocaulus vivparus (lungworm)

Muellerius capillaris (goat lung worm)

Fasciola hepatica (liver fluke)

Haemonchus contortus (barber pole worm)


Ostertagia circumcincta (brown stomach worm)

Trichostrongylus spp. (hair worm)

Nematodirus spp (thin-necked intestinal worm)

Oesophagostomum columbianum (nodular worm)


Strongyloides papillosus (common thread worm)

Parelaphostrongylus tenuis (brain or meningeal worm)

Trichuris ovis (whipworm)

Moniezia expansa (tapeworm)


+ نوشته شده در  سه شنبه بیست و پنجم مرداد 1390ساعت 9:13 AM  توسط امین احمدی   | 


Goat owners mount a significant effort to eliminate infections of parasites which can cause harm and misery to their goats. If, for only a short time, the worry over parasites can be set aside in favor of a closer look at their life history, interesting, even mysterious discoveries can be learned.


The relationship of internal parasites to the host is often complex. Larvae can go through phases in which one stage bears no resemblence to another. Some parasites require more than one different species of host, while others can alternate between free-living or parasitic forms.
Adults of internal parasites must live and take nourishment in some organ of the host and then find a way to infect other hosts. With few exceptions, the egg once laid, exits the host, hatches in the often hostile environment, then the juvenile larvae must find a way to get inside another host to develop to the adult stage.

Direct Life Cycles

The life cycle of a nematode worm, such as Haemonchus contortus which causes so much trouble for goats and their owners, is an example of a simple, direct type of life cycle which requires only one host.

The adult, female worm lays eggs which leave the goat's intestinal tract with the feces. The first-stage larvae hatch out and feed on soil microbes and bacteria. When the first-stage larva grows large enough it matures into the second stage. The somewhat rigid, protective outside membrane called the cuticle must be shed so the larva can grow. The second stage continues to eat and when the time comes to molt to the third stage, the larva does not shed its protective cuticle, but retains it. The third stage larva then has two protective cuticles called a sheath which helps it survive in the environment. However, the two membranes completely encloses the larva which prevents its ability to eat.

The third-stage larvae spend the next few months working a short way up moist grass every morning, waiting patiently for a goat to come along and eat it.When the sun gets too hot and the grass dries out, the larvae crawl back down to the cooler, moist surface to wait for another day.

If the larva is lucky enough to be ingested by the goat, the third cuticle is shed with the help of digestive enzymes in the rumen allowing the fourth stage larva to emerge by the time it reaches the fourth stomach. After feeding at the lining of the stomach wall or small intestine for a short time, the fourth stage develops into the adult stage and begins mating and laying eggs shortly after.

Another type of nematode parasite which usually does not cause the goat much trouble has a more fascinating life cycle. Males and females of Strongyloides papillosus, the intestinal thread worm, live freely in the soil almost everywhere in the world. They feed, mate and lay thin-shelled eggs which produce more free-living worms. This free-living worm also goes through four larval stages, but does not require a animal host.


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